Inflammatory illness of the skin, acnes Vulgaris is caused by changes in the pilosebaceous units of the skin’s epidermis (skin structures consisting of a hair follicle and its associated sebaceous gland). Acne lesions are frequently referred to as pimples, spots, or zits, depending on their appearance. acne treatment, acne scars, acne Symptoms, acne cream
Puberty is a frequent time for the illness, particularly in Western civilizations, and it is thought to be caused by a greater genetic susceptibility. As a result of normal amounts of the male hormone testosterone, it is seen as an aberrant reaction. After one enters their early twenties, the majority of individuals see a decline in their immune response, and acne tends to vanish, or at the very least lessen.
No one can anticipate how long it will take for acne to completely go, and some people will continue to suffer from it decades later, far into their thirties, forty-five, and even later in their lives. Ac-ne affects a substantial majority of the population at some point in their lives.
Ac-ne vulgaris, or “common ac-ne,” is the most common kind of acne and is the most prevalent type of ac-ne. Sebaceous glands secrete excessive amounts of oils, which interact with naturally existing dead skin cells to clog hair follicles, causing them to get clogged. There also seems to be a problem with the keratinization process in the skin in certain cases, which results in abnormal shedding of the skin lining the pores in the skin.
Under the blocked pore, oil discharges accumulate, creating an ideal habitat for the skin germs Propionibacterium ac-nes to flourish uncontrollably. As a result, the skin becomes inflamed, resulting in an apparent lesion. The face, chest, back, shoulders, and upper arms are the most often afflicted areas of the body.
Acne lesions include comedones, papules, pustules, nodules, and inflammatory cysts, to name a few examples. These are the most inflammatory forms of pus-filled or crimson pimples or even sensitive swellings that look like boils.
Non-inflamed sebaceous cysts,’ more formally known as epidermoid cysts, may develop in connection with acne or on their own, although they are not a common occurrence. They are also not a permanent characteristic of the skin. Following the elimination of ac-ne lesions, it is possible that large and ugly scars may remain.
Except for the physical impacts of scarring, most of the psychological consequences include low self-esteem and despair. Ac-ne frequently emerges throughout adolescence, when individuals are already at their most uneasy in their social situations.
Acne has a variety of causes.
It is not completely understood why some individuals get acne while others do not. It is known to be inherited to some extent. It is recognized that ac-ne is caused by a number of variables, including:
Menstrual cycles and puberty, for example, are examples of hormonal activity.
Stress causes an increase in hormone production from the adrenal (stress) glands, which causes the body to become more agitated.
Sebaceous glands are overactive as a result of the three hormone sources mentioned above.
Dead skin cells have accumulated on the skin’s surface.
Pores are clogged with bacteria to which the body has a ‘allergic reaction’.
An irritant or scratching action of any kind on the skin will cause inflammation.
The use of anabolic steroids is prohibited.
Anything that contains halogens (iodides, chlorides, bromides), lithium, barbiturates, androgens, or any combination of these.
High quantities of chlorine compounds, especially chlorinated dioxins, may produce severe, long-lasting acne, known as Chlorac-ne, when exposed to them in high concentrations.
Most emphasis has traditionally been placed on the overproduction of sebum caused by hormones, which is considered to be the primary contributory reason to acne. Increasingly lately, the narrowing of the follicular channel has received more attention as a second major contributing cause to the development of ac-ne.
Some researchers believe that aberrant shedding of cells that line the follicle, abnormal cell binding (“hyperkeratinization”) inside the follicle, and water retention in the skin (which causes the skin to expand and therefore push the follicles shut) are all essential factors. The hormone testosterone, as well as the male hormones dihydrotestosterone (DHT) and dehydroepiandrosterone sulfate (DHEAS), as well as insulin-like growth factor 1 (IGF1), have all been associated with ac-ne (IGF-I). Furthermore, it has been shown that ac-ne-prone skin is insulin resistant.
Acne vulgaris development in the latter years is rare, despite the fact that this is the age group for Rosacea, which may have comparable characteristics to acne vulgaris. For those over the age of 50, true ac-ne vulgaris is often associated with an underlying disease such as pregnancy and hormonal problems such as polycystic ovarian syndrome or the unusual Cushing’s syndrome.
Misconceptions concerning the underlying reasons
The following are some common myths and rumors regarding what causes the illness and what does not.
Diet. One erroneous research claimed that ac-ne was exacerbated by foods such as chocolate, french fries, potato chips, and sugar, among other things. According to a recent survey of scientific literature, there is no definitive answer. It is generally agreed upon by health specialists that ac-ne patients should experiment with their diets and avoid eating certain foods if they discover that they are making their ac-ne worse. An epidemiological relationship between milk intake and acne, namely skimmed milk, was found in recent research based on a survey of 47,335 women, according to the findings.
According to the researchers, hormones (such as bovine IGF-I) found in cow milk may be responsible for the relationship, although this has not been shown conclusively. For its part, seafood may contain high quantities of iodine, but not enough to produce an ac-ne breakout. People who are prone to ac-ne, on the other hand, may wish to avoid consuming excessive amounts of iodine-rich foods. According to some reports, a diet consisting mostly of refined sugars may be associated with ac-ne-prone skin.
It is hypothesized that the stunning lack of acne in non-westernized communities may be attributed to the low glycemic index of the meals of these tribes’ members. In order to determine whether a reduced consumption of high-glycemic foods (such as soft drinks, sweets, and white bread) can significantly alleviate ac-ne, additional research is required. However, the consumption of high-glycemic foods should be kept to a bare minimum for the sake of general health.
Personal hygiene is not up to par. Ac-ne is not caused by dirt in the skin. This misunderstanding is most likely caused by the fact that ac-ne is associated with skin illnesses. In reality, the clogs that cause ac-ne are located deep inside the tiny follicle channel, where they are hard to remove with soap and water. These plugs are generated by the cells and sebum produced by the body in the affected area.
They are the same germs that are usually present on the skin and are responsible for the infection. Regular washing of the skin may help to minimize, but not completely eliminate, acne for a specific person, and there is very little variance across people owing to cleanliness. The use of anything other than the most delicate washing products may actually aggravate existing lesions and even stimulate the development of new ones by harming or overdrying the skin.
Sex. Most people believe that either celibacy or masturbation causes acne and that sexual contact may help to treat it. This is a common misconception. The existence of any of these claims is not supported by any scientific evidence at the present time. However, it is true that anger and stress have an impact on hormone levels and, as a result, on body oil production. It is now being investigated whether or not any increases in oil production caused by stress are sufficient to cause ac-ne to appear.